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Cancer Treatment Reviews
Volume 29, Issue 1
, Pages 31-44
, February 2003
Recent advances in the treatment and understanding of childhood acute lymphoblastic leukaemia
References
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Tandem application of flow cytometry and polymerase chain reaction for comprehensive detection of minimal residual disease in childhood acute lymphoblastic leukaemia.
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Long-term results of four consecutive trials in childhood ALL performed by the ALL-BFM study group from 1981 to 1995. Berlin–Frankfurt–Muenster.
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Co-operative study group for childhood acute lymphoblastic leukaemia (COALL): long-term follow-up of trials 82, 85, 89 and 92.
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Long-term follow-up of dutch childhood leukaemia study group (DCLSG) protocols for children with acute lymphoblastic leukaemia, 1984–1991.
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Six months of maintenance chemotherapy after intensified treatment for acute lymphoblastic leukaemia of childhood.
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Possible implication of thiopurine S-methyltransferase in occurrence of infectious episodes during maintenance therapy for childhood lymphoblastic leukaemia with mercaptopurine.
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Duration and intensity of maintenance chemotherapy in acute lymphoblastic leukaemia: overview of 42 trials involving 12,000 randomised children. Childhood ALL Collaborative Group. Lancet 1996;347:1783–1788
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Osteonecrosis as a complication of treating acute lymphoblastic leukaemia in children: a report from the children’s cancer group.
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Magnetic resonance imaging detection of avascular necrosis of the bone in children receiving intensive prednisone therapy for acute lymphoblastic leukaemia or non-Hodgkin lymphoma.
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Bony morbidity in children treated for acute lymphoblastic leukaemia.
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Long-term follow-up of the United Kingdom medical research council protocols for childhood acute lymphoblastic leukaemia, 1980–1997. Medical research council childhood leukaemia working party.
Leukaemia. 2000;14:2307–2320
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Secondary brain tumors in children treated for acute lymphoblastic leukaemia at St. Jude children’s research hospital.
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Pilot studies of species-specific chemotherapy of childhood acute lymphoblastic leukaemia using genotype and immunophenotype.
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Long-term results of three randomized trials (58831, 58832, 58881) in childhood acute lymphoblastic leukaemia: a CLCG-EORTC report. Children leukaemia cooperative group.
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- Outcomes of growth hormone replacement therapy in survivors of childhood acute lymphoblastic leukaemia. J. Clin. Oncol. 2002;20:2959–2964
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Prognostic significance of cerebrospinal fluid (CSF) lymphoblasts (LB) at diagnosis (dx) in children with acute lymphoblastic leukaemia (ALL) (Meeting abstract).
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Low numbers of CSF blasts at diagnosis do not predict for the development of CNS leukaemia in children with intermediate-risk acute lymphoblastic leukaemia: a children’s cancer group report.
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Effect of initial central nervous system (CNS) status on event-free (EFS) in children and adolescents with acute lymphoblastic leukaemia (ALL).
Med. Pediatr. Oncol. 2002;39:277;
(abstract)
- . Quantitative assessment of cerebral atrophy during and after treatment in children with acute lymphoblastic leukaemia. Invest. Radiol. 1996;31:749–754
- . Chemotherapy for acute lymphoblastic leukaemia may cause subtle changes of the spinal cord detectable by somatosensory evoked potentials. Med. Pediatr. Oncol. 1997;28:41–47
- . Visual and verbal short-term memory deficits in childhood leukaemia survivors after intrathecal chemotherapy. J. Pediatr. Psychol. 1997;22:861–870
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Howard SC, Gajjar A, Cheng C, Kritchevsky SB, Somes GW, Harrison PL, et al. Traumatic lumbar puncture in childhood acute lymphoblastic leukaemia. J Am Med Assoc 2002; in press
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Safety of lumbar puncture for children with acute lymphoblastic leukaemia and thrombocytopenia.
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Sequential changes in platelet function and coagulation in leukaemic children treated with l-asparaginase, prednisone, and vincristine.
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- . Drug resistance in haematologic malignancies. Curr. Opin. Oncol. 2001;13:463–469
- The ABC transporter Bcrp1/ABCG2 is expressed in a wide variety of stem cells and is a molecular determinant of the side-population phenotype. Nat. Med. 2001;7:1028–1034
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Gene therapy to protect haematopoietic cells from cytotoxic cancer drugs.
Nat. Rev. Cancer. 2002;2:431–441
- Efficacy and safety of a specific inhibitor of the BCR-ABL tyrosine kinase in chronic myeloid leukaemia. N. Engl. J. Med. 2001;344:1031–1037
- Haematologic and cytogenetic responses to imatinib mesylate in chronic myelogenous leukaemia. N. Engl. J. Med. 2002;346:645–652
- Imatinib induces durable haematologic and cytogenetic responses in patients with accelerated phase chronic myeloid leukaemia: results of a phase 2 study. Blood. 2002;99:1928–1937
- Imatinib induces haematologic and cytogenetic responses in patients with chronic myelogenous leukaemia in myeloid blast crisis: results of a phase II study. Blood. 2002;99:3530–3539
- Activity of a specific inhibitor of the BCR-ABL tyrosine kinase in the blast crisis of chronic myeloid leukaemia and acute lymphoblastic leukaemia with the Philadelphia chromosome. N. Engl. J. Med. 2001;344:1038–1042
- A phase 2 study of imatinib in patients with relapsed or refractory Philadelphia chromosome-positive acute lymphoid leukaemias. Blood. 2002;100:1965–1971
- Inhibition of mutant FLT3 receptors in leukaemia cells by the small molecule tyrosine kinase inhibitor PKC412. Cancer Cell. 2002;1:433–443
- CT53518, a novel selective FLT3 antagonist for the treatment of acute myelogenous leukaemia (AML). Cancer Cell. 2002;1:421–432
- A FLT3-targeted tyrosine kinase inhibitor is cytotoxic to leukaemia cells in vitro and in vivo. Blood. 2002;99:3885–3891
PII: S0305-7372(02)00106-8
doi: 10.1016/S0305-7372(02)00106-8
© 2003 Elsevier Science Ltd. All rights reserved.
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Cancer Treatment Reviews
Volume 29, Issue 1
, Pages 31-44
, February 2003
